横河電機株式会社
横河ソリューションサービス株式会社

論文リスト : CQ1

[1] D. W. Cleveland et al., “TRIP13 and APC15 drive mitotic exit by turnover of interphase- and unattached kinetochore-produced MCC,” Nat. Commun., vol. 9, no. 1, p. 4354, 2018.

[2] F. Gasset-Rosa et al., “Cytoplasmic TDP-43 De-mixing Independent of Stress Granules Drives Inhibition of Nuclear Import, Loss of Nuclear TDP-43, and Cell Death,” Neuron, Mar. 2019.

[3] T. Hoshiba et al., “Maintenance of Cartilaginous Gene Expression of Serially Subcultured Chondrocytes on Poly(2-Methoxyethyl Acrylate) Analogous Polymers,” Macromol. Biosci., vol. 17, no. 12, p. 1700297, Jun. 2017.

[4] J. Hwang et al., “Synthesis and Characterization of Functional Nanofilm-Coated Live Immune Cells,” ACS Appl. Mater. Interfaces, vol. 10, no. 21, pp. 17685–17692, 2018.

[5] V. G. Kartzev et al., “Discovery and optimization of cardenolides inhibiting HSF1 activation in human colon HCT-116 cancer cells,” Oncotarget, vol. 9, no. 43, 2018.

[6] D. H. Kim et al., “The AAA+ ATPase TRIP13 remodels HORMA domains through N‐terminal engagement and unfolding,” EMBO J., vol. 36, no. 16, pp. 2419–2434, 2017.

[7] S. W. Kim et al., “Mutual Destruction of Deep Lung Tumor Tissues by Nanodrug-Conjugated Stealth Mesenchymal Stem Cells,” Adv. Sci., vol. 5, no. 5, p. 1700860, Jun. 2018.

[8] T. Kimura et al., “Lipophagy maintains energy homeostasis in the kidney proximal tubule during prolonged starvation,” Autophagy , vol. 13, no. 10, pp. 1629–1647, 2017.

[9] H. Komura et al., “Alzheimer Aβ Assemblies Accumulate in Excitatory Neurons upon Proteasome Inhibition and Kill Nearby NAKα3 Neurons by Secretion,” iScience, 2019.

[10] Y. S. Lee and H. S. Jun, “Glucagon-like peptide-1 receptor agonist and glucagon increase glucose-stimulated insulin secretion in beta cells via distinct adenylyl cyclases,” Int. J. Med. Sci., vol. 15, no. 6, pp. 603–609, 2018.

[11] F. Louis, S. Kitano, J. F. Mano, and M. Matsusaki, “3D collagen microfibers stimulate the functionality of preadipocytes and maintain the phenotype of mature adipocytes for long term cultures,” Acta Biomater., vol. 84, pp. 194–207, Jan. 2019.

[12] F. Meitinger et al., “53BP1 and USP28 mediate p53 activation and G1 arrest after centrosome loss or extended mitotic duration,” J. Cell Biol., vol. 214, no. 2, pp. 155–166, 2016.

[13] M. H. Mosa et al., “Dynamic Formation of Microvillus Inclusions During Enterocyte Differentiation in Munc18-2–Deficient Intestinal Organoids,” Cmgh, vol. 6, no. 4, pp. 477-493.e1, Aug. 2018.

[14] K. Nanki et al., “Divergent Routes toward Wnt and R-spondin Niche Independency during Human Gastric Carcinogenesis,” Cell, vol. 174, no. 4, pp. 856-869.e17, Aug. 2018.

[15] S. Santaguida et al., “Chromosome Mis-segregation Generates Cell-Cycle-Arrested Cells with Complex Karyotypes that Are Eliminated by the Immune System,” Dev. Cell, vol. 41, no. 6, pp. 638-651.e5, 2017.

[16] H. Seo et al., “A β1-tubulin–based megakaryocyte maturation reporter system identifies novel drugs that promote platelet production,” Blood Adv., vol. 2, no. 17, pp. 2262–2272, Sep. 2018.

[17] Y. Shimada et al., “FF-10502, an Antimetabolite with Novel Activity on Dormant Cells, Is Superior to Gemcitabine for Targeting Pancreatic Cancer Cells,” J. Pharmacol. Exp. Ther., vol. 366, no. 1, pp. 125–135, 2018.

[18] N. Sunamura, S. Iwashita, K. Enomoto, T. Kadoshima, and F. Isono, “Loss of the fragile X mental retardation protein causes aberrant differentiation in human neural progenitor cells,” Sci. Rep., vol. 8, no. 1, p. 11585, Dec. 2018.

[19] M. Tanaka et al., “Adhesion-based simple capture and recovery of circulating tumor cells using a blood-compatible and thermo-responsive polymer-coated substrate,” RSC Adv., vol. 6, no. 92, pp. 89103–89112, 2016.

[20] C. Zhang et al., “Mimicking Pathogenic Invasion with the Complexes of Au22(SG)18-Engineered Assemblies and Folic Acid,” ACS Nano, vol. 12, no. 5, pp. 4408–4418, 2018.

論文検索サイトへのリンク

 

 


公式アカウント

ライフサイエンスに関する最新情報を定期的に配信しています。是非フォローをお願いいたします。

  Follow us Share our application
•Twitter @Yokogawa_LS Share on Twitter
•Facebook Yokogawa Life Science Share on Facebook
•LinkedIn Yokogawa Life Science Share on LinkedIn

横河電機株式会社公式アカウントリスト

Social Media Account List


関連製品&ソリューション

  • ハイコンテントアナリシス CellVoyager

    CellVoyagerシリーズは生きた細胞の様々な反応を高速かつ詳細に調べることにより、新薬開発や化合物評価、細胞機能解明などの効率を上げるハイスループットハイコンテントアナリシスシステムです。最先端のライフサイエンス研究のニーズに応え、常に進化を続けています。

    さらに見る

トップ